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Page 1: EKG 10

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EKG patologic

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Hipertrofie atriala dreapta

• P inalta (>2,5 mm) si ascutita in derivatii

inferioare DII, DIII, aVF (p pulmonar) sau p

difazic in V1/V2

• HTP

• stenoza/insuf tricuspidiana

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Hipertrofie atriala stanga

• Unda p larga >0,12 sec, bifida in DI, DIII si aVL

(p mitral) sau difazica in V1

• Stenoza/ insuf Mi

• Stenoza/ insuf Ao

• Insuf VS

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Hipetrofii ventriculare

• Alterarea depolarizarii

- cresterea amplitudinii si duratei undelor R

- modificari ale axului inimii

• Alterarea repolarizarii

- Modificari secundare ale fazei terminale(segment ST si unda T)

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Modificari de faza terminala

• In opozitie fata de deflexiunea majora a

complexului QRS:

 – In derivatiile directe (de ex pt VS V5,V6 si pentru

VD V1, V2)- T inversat si ST subdenivelat

 – In derivatiile indirecte (VS- V1, V2 si VD- V5, V6)-

ST supradenivelat si T pozitiv, simetric

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Hipertrofie ventriculara dreapta

- R>S in V1- V2 SAU R V1>7 mm

- S adanc in V5,V6

- Deflexiune intrinsecoida > 0.035-0.055 s in V1

- R V1 + S V5/6> 10.5 mm

- qrS in derivatiile drepte

- Modificari de faza terminala- subdenivelare ST si inversare T in V1,V2

- Ax deviat la dreapta

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Cauze HVD

• B pulmonare cronice

• Stenoza mitrala

Stenoza pulmonara

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Hipertrofie ventriculara stanga

• In derivatii frontale – RDI + S DIII >25 mm

 – R in derivatii inf (DII, DIII, aVF)>20 mm

 – R aVR> 13 mm

•In derivatii orizontale – Indice SOKOLOV-LYON

S V1+R V5/V6> 35 mm

- S maxim V1/V2 > 26 mm

- R maxim V5/V6 >26 mm

• Faza terminala- opozitie de faza in V5,V6,DI si aVL• Ax QRS normal dar deviat spre 0-10 grade; HVS f severa-

spre – 30 grade

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Cauze HVS

• HTA

Stenoza Ao• Insuf Ao

• Insuf Mi

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• SOKOLOV LYON INDEX= R in V5/ V6 + S in V1 >35 mm

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Hipokalemie

• Modif EKG- cand valoarea este sub 3mEq/l

Amplitudine T scazuta• Subdenivelare ST

• Unda U

• QT scurt• Tulburari de ritm

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Hiperkalemie

• T amplu

• Aplatizare p pana la disparitie

QRS larg• Aritmii

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TULBURARI DE RITM

Aritmii sinusale

- Tahicardie sinusala

- Bradicardie sinusala

- Aritmie respiratorie

- Pauza sinusala

Aritmii atriale- Extrasistola atriala

- Fibrilatie atriala

- Flutter atrial

- Tahicardie paroxistica supraventriculara

Aritmii ventriculare- Extrasistola ventriculara

- Tahicardie ventriculara

- Fibrilatie ventriculara

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Tahicardie sinusalaMecanism:

descarcare crescuta a NS

Ritm: sinusal regulatFrecventa: >100 bpm

- la adulţi nu depăşeste 140-180 bpm

- la copii 200-220 bpm

Unde P: normale, uniforme; daca ritmul este crescut unda P se poate pierde in unda T

Interval PR: normal (0.12 – 0.20 sec), dar poate scadea cu ↑ ratei

QRS: normal (0.06 – 0.10 sec)

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Tahicardie sinusala

•   Cauze:

- Fiziologice: exercitii ,anxietate, durere,

- Patologice: febra, anemie, hipovolemie, hipoxie, insuf 

cardiaca

- Endocrine: tireotoxicoze- Farmaceutice: adrenalina eliberata in feocromocitom;

salbutamol (medicamente simpatomimetice), alcoolul,

cafeina

- Poate fi primul semn al Insuf Ventric Stg

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Bradicardie sinusalaMecanism: descarcare scazuta a NSA

Frecventa: < 60 bpm

Ritm: regulat

Unde P: normale, uniforme, urmate de compl qRS

Interval PR: normal (0.12 – 0.20 sec),

QRS: normal (0.06 – 0.10 sec)

- Bradicardia sinusala este normala in timpul somnului si la

persoanele cu tonus vagal crescut, cum ar fi atletii si adultii

tineri sanatosi.

- Cea mai obisnuita cauza patologica este IMA

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Aritmie respiratorie

• Aritmie fiziologica

• Cresterea frecventei cardiace in inspir si

scaderea in expir (expirul stimuleaza nc

ambiguu + nv X scade frecventa cardiaca)

• Oprirea voluntara a respiratiei duce la

disparitia aritmiei

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Pauza sinusala

• Lipsa generarii impulsurilor cu absenta

undelor p

• Durata intervalului fara unde p nu se coreleaza

cu ritmul de baza

• Bradicardie severa

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Extrasistola atriala

• QRS prematur

• Unda p a complexului ES are morfologie diferita

• Urmata de o pauza necompensatorie

QRS de durata normala- conducere pe cai normale

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Fibrilatia atriala

• Focare ectopice in atrii

• absenta undelor p- inlocuite de mici oscilatii

ale liniei izoelectrice numite unde f de

fibrilatie, cel mai bine vizibile in V1/V2

• frecventa atriala este de 400-600/min

• QRS inguste, dar complet neregulate

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Flutter atrial

• Focare ectopice multiple atriale cu mecanism

de reintrare

• Fara unde p, inlocuite cu unde F de flutter in

dinti de fierastrau

• Frecventa de 250-350/min

• Frecventa ventriculara este de obicei la

 jumatate din cea atriala

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Tahicardie paroxistica

supraventriculara

• Frecventa cardiaca de 150-250/min

• Succesiune de minim 5-6 ESA

P prezente, dar de morfo diferita (alt focar)• QRS de aspect normal

• Se opreste cu manevre vagale (masaj

carotidian, compresie glob ocular)

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Extrasistola ventriculara

• QRS prematur, dar fara unda p ce il precede

• Pauza compensatorie

• QRS larg>0,12 s

Mai mult de 3 ESV= tahicardie ventriculara

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Tahicardie ventriculara

• Trei sau mai multe ESV consecutive

• Frecventa de >100/min

• QRS pot fi monomorfe sau polimorfe

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Fibrilatie ventriculara= stop cardiac

(contractii ineficiente)

• Unde rapide neregulate cu frecventa de 130-

300/min

• Complexe QRS aberante- largi si deformate

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Anomalii de conducere

• Bloc SA

• Bloc AV

• Bloc de ramura dr/stg

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Bloc AV

• Gr I- pR> 0,21 s

• Gr II

 – A. Mobitz 1- cu perioade Luciani Wenckebach

 – B. Mobitz 2

• Gr III- complet

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Bloc AV I= PQ/PR > 0.21 s

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Bloc AV II.1- Mobitz 1 cu perioade

Luciani- Wenckebach• Alungire progresiva a intervalului PR, cu fiecare ciclu succesiv, pana cand

o unda p este complet blocata

• Distanta dintre 2 unde p blocate se numeste per LW

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BAV II.2- Mobitz 2

• Interval PR normal cu blocarea brusca a

conducerii undei p catre ventriculi

• Gradele blocarii impulsurilor pot fi de 2/1;

3/1; 4/1

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BAV III- complet

• Blocarea completa a conducerii AV

• frecventa atriala- ritm sinusal/alt ritm atrial

• Ventriculii- ritm de scapare cu frecventa de 30-40/min

• Disociatie completa atrioventriculara

• Implant de pacemaker permanent

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Bloc de ramura

• stanga- QRS larg V5, V6

• dreapta- QRS larg V1, V2

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BRD

• Ritmul e generat deasupra ventriculilor

• QRS >100 ms- bloc incomplet

• QRS> 120 ms bloc complet

• Unda R terminala in V1 R, rR', rsR', rSR' or qR

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BRS

• Ritm generat deasupra ventriculilor

• QRS≥ 120 ms

• QS / rS in V1

• RsR‘ in V6.

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SINDROAME DE PREEXCITATIE

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• CONDUCERE DIRECTA A IMPULSULUI DE LA A-

V PRIN FASCICULE ABERANTE- SUNTAREA NAV

• TIPURI

Wolf- Parkinson- White (WPW)

Lown- Ganong- Levine (LGL)

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Wolf- Parkinson- White (WPW)

• FASCICUL KENT- PR< 0,12, UNDA DELTA, QRS >

0,1s

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Lown- Ganong- Levine (LGL)

- INITIAL S-A CREZUT IN EXISTENTA F JAMES- de

fapt NAV conduce mai rapid- PR< 0,12, fara

unda Delta- problema este intranodala- fara

unda de sumatie

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Tulburari de perfuzie

• ischemie- cea mai usoara- este reversibila siafecteaza repolarizarea- subdenivelare ST +/- Tnegative, simetrice

leziune- stadiu intermediar, greu reversibil,produce intarziere de repolarizare- pe EKGmodificari de segment ST: supradenivelare de ST

• necroza= moartea miocardului- nu este

reversibila- pe EKG= unda Q de necroza (cuamplitudine >1/4 R si durata >0,04 s, in derivatiilecorespunzatoare

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IM acut

Stadii

1.Unda T pozitiva, ampla, larga- T hiperacut- in primeleore de la debut

2.Supradenivelare ST, initial concava, apoi convexa- undaPardee- se reduce progresiv in evolutie pana ajunge lalin izoel in 1-2 sapt de la debut=leziune

3.Unda T devine negativa, ascutita, simetrica in 1-2 zile,maxim in primele 2 sapt=ischemie

3.Unda Q larga si adanca dupa 8-10 ore de la debut(durata>0.04s si ampl>1/4 din R adiacent)=necroza

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Leziune

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Acute myocardial infarction

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Inferior infarction=Q in D2, D3, aVF